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acetaminophen overdose napqi glutathione mechanism authoritative source Translational biomarkers of acetaminophen-induced acute liver injury | Archives of Toxicology nad-plus
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For example, injured proximal tubule cells undergo cell cycle arrest, change the substrate utilization for ATP generation from fatty acids to glucose ( β -oxidation to glycolysis), and accumulate lipid droplets in response to stress
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These findings indicated that high glucose induces PSC activation via the TGFβ signalling pathway
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