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[DOI] [PMC free article] [PubMed] [Google Scholar] 348.Liu L., Bai X., Martikainen M.-V., Kårlund A., Roponen M., Xu W., Hu G., Tasciotti E., Lehto V.-P
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These include regulation of mechanisms controlling reactive oxygen species (ROS), which cause macromolecular damage 16,17 , mitochondrial turnover or mitophagy 18 , and the mitochondrial unfolded protein response (UPR mt ), in which mitochondrial damage can be prevented by the removal of aggregated or unfolded proteins 19
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