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Where the evidence actually stands To counsel a patient honestly about TB-500, a clinician needs a vocabulary for how strong the evidence is — not just whether something has been "studied." A useful way to frame it is to think of TB-500 as a "tier-four" compound : strong animal data, a mechanistically plausible rationale, and real anecdotal and case-report interest, but limited or no published human randomized controlled trials on the marketed product itself

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This can be readily explained by the observation [22], that complete disruption of MCT activity in LS174T cells by a combination of genetic and pharmacological means results in a more than six-fold increase in oxidative phosphorylation, which leads in turn to the increased generation of mitochondrial reactive oxygen species (ROS) and, consequently, an increase in cellular content of GSSG