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Ferroptosis in viral infection: the unexplored possibility | Acta Pharmacologica Sinica
Moreover, proteasome dysfunction may further potentiate ER stress and ferroptotic pathways by allowing the persistence of oxidised and lipid-peroxidation–derived adducts, thereby integrating UPS failure into a broader cascade of proteotoxic and oxidative injury [20,40]
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Is oxidative damage the fundamental pathogenic mechanism of Alzheimer's and other neurodegenerative diseases
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