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Interestingly, despite the activation of caspases in late stages of parthanatos, caspase inhibitors cannot inhibit the parthanatos process except for blocking PARP1 activity, confirming that PARP1-mediated cell death is caspase-independent [137, 139]

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Under homeostatic states, glutamine metabolism supports the basal synthesis of purines and nucleotides and contributes to the production of glutamate, aspartate, and lactate