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A Literature Review of Glutathione Therapy in Ameliorating Hepatic Dysfunction in Non-Alcoholic Fatty Liver Disease - PMC
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Bioinformatic analysis of the molecular function pathways revealed dysregulation of proteins that interact with receptor tyrosine kinase, cadherin, and cell adhesion molecules, which disrupt signaling mechanisms related to cell survival, growth, inflammation, and structural integrity, RNA binding, and protein homodimerization, and which are important for gene expression regulation, protein-protein interactions, and the cellular stress response (Fig
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