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autism glutathione genes Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with personal care
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autism glutathione genes Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with personal care
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autism glutathione genes Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with personal care
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autism glutathione genes Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with personal care
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In a previous study, mitochondrial fusion protein-2 (MFN2) enhanced mitochondria-endoplasmic reticulum (ER) contact and fostered inflow of mitochondrial Ca 2+ required for efficient mitochondrial metabolism by interacting with the ER-embedded Ca-ATPase SERCA2, thereby preventing mitochondrial Ca 2+ overaccumulation and apoptosis
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Age group:Adults
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autism glutathione genes Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with personal care
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