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glutathione peroxidase 4 mouse GPX4-independent ferroptosis—a new strategy in disease's therapy concern_Dark Circles
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Thus, ROS may also contribute and accelerate the insulin resistance development in insulin-targeted organs of the over nutrition and the excess salt individuals
homozygous mice expressing catalytically inactive GPx4 variants (Sec46Ala/Ser mutant) do not survive beyond the 7th day of embryogenesis
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