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In the case of ALS, mutant forms of copper-zinc superoxide dismutase (SOD1), which are collectively responsible for approximately 20% of cases of familial ALS, accumulate at mitochondria and trigger a shift in the redox state of these organelles [9]
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Specification:20 mg
Quality Assurance HPLC Verification: Each peptide undergoes rigorous high-performance liquid chromatography to confirm analytical purity prior to lyophilisation
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