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Its central action in research is competitively blocking NNMT's active site, which does two coupled things: it stops the methylation of nicotinamide (a NAD+ precursor) into 1-methylnicotinamide, and it spares S-adenosylmethionine (SAM), the methyl donor the reaction consumes
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Strength:10mg
It is not prepared or labeled for people or animals and is not a stand-in for any approved or investigational drug
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