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Overexpressing FSP1 in cells lacking GPX4 expression was shown to significantly reduce specific phospholipid peroxidation products compared to control cells [15, 16]

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Interestingly, because apoptosis is used by the host as an antiviral defense strategy in order to eliminate virus, HSV-1 encodes numerous viral determinants for evading it (Figure 2A), which overall may favor virus persistence in the host by remaining in a viable substrate as a long-term niche for latency, which could produce chronic inflammation due to recurrent asymptomatic reactivations (Perng et al., 2000)