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Unlike the positive influence of most indole derivatives in the gut, IAA, indoxyl sulfate, and indoxyl-β-D-glucuronide act in cells of the cardiovascular system as pathogenic agents that, via the AHR, induce the transcription of proinflammatory cytokines, apoptosis, and oxidative stress
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10.1007/s11064-013-1141-x Neurochem
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