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Summary and perspectives This review highlights the pivotal role of amino acid metabolism in the pathogenesis of pulmonary fibrosis, with a focus on how dysregulated metabolic pathways—particularly those involving proline, arginine, glutamine, and tryptophan—drive collagen deposition, fibroblast activation, and disease progression
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According to a hospital‐based case–control study, Th17/Treg balance is impacted by vitamin A deficiency, which is a poor prognostic factor in SLE patients
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