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FIGURE 1 As a counterbalanced pathway, the nuclear factor erythroid 2-related factor 2 (Nrf2) is often activated by transient oxidative or electrophilic stress and moves into the nucleus of VSMC, for example, and increases the expression of SOD, CAT, glutathione peroxidase (GPx), NAD(P)H quinone oxidoreductase 1 (NQO1) and heme oxygenase (HO)-1, thereby decreasing the availability of RONs (Ch et al., 2018)